| Oct | 2008 | |
| 7 | Georgetown - SDP | |
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Muller cells are living optical fibers in the vertebrate retina
Proc Natl Acad Sci USA. 2007 May 15; 104(20): 8287-92.
Although biological cells are mostly transparent, they are phase objects that differ in shape and refractive index. Any image
that is projected through layers of randomly oriented cells will normally be distorted by refraction, reflecting and scattering.
Counterintuitively, the retina of the vertebrate eye is inverted with respect to its optical function and light must pass
through several tissue layers before reaching the light-detecting photoreceptor cells. Here we report on the specific optical
properties of glial cells present in the retina, which may contribute to optimize this apparently unfavorable situation. We
investigated intact retinal tissue and individual Muller cells, which are radial glial cells spanning the entire retinal thickness.
Muller cells have an extended funnel shape, a higher refractive index than their surrounding tissue, and are oriented along
the direction of light propagation. Transmission and reflection confocal microscopy of retinal tissue in vitro and in vivo
showed that these cells provide a low-scattering passage for light from the retinal surface to the photoreceptor cells. Using
modified dual-beam laser trap we could also demonstrate that individual Muller cells act as optical fibers. Furthermore, their
parallel array in the retina is reminiscent of fiber optic plates used for low-distortion image transfer. Thus, Muller cells
seem to mediate the image transfer through the vertebrate retina with minimal distortion and low loss. This finding elucidates
a fundamental feature of the inverted retina as an optical system and ascribes a new function to glial cells.
Zhao HW, Ross AP, Christian SL, Buchholz JN, Drew KL
Decreased NR1 phosphorylation and decreased NMDAR function in hibernating Arctic ground squirrels.
J Neurosci Res. 2006 Aug 1; 84(2): 291-8.
Heterothermic mammals such as ground squirrels tolerate ischemia and N-methyl-D-aspartate (NMDA) better than homeothermic
mammals such as rats both in vivo and in vitro, and this tolerance is enhanced in the hibernating state. However, the cellular
mechanisms underlying this tolerance remain unclear. NMDA receptors (NMDAR) play a key role in excitotoxicity. The purpose
of the current study was therefore to test the hypothesis that NMDAR are down-regulated in hibernating Arctic ground squirrels
(hAGS; Spermophilus parryii). To address this hypothesis, we used Western blot analysis to investigate NMDAR phosphorylation,
an activator of NMDAR function, and internalization in naive hippocampal tissue from hAGS, interbout euthermic AGS (ibeAGS),
and rats. Furthermore, we used fura-2 calcium imaging to examine NMDAR function in cultured hippocampal slices from hAGS,
ibeAGS, and rats. We report that phosphorylation of the NMDAR1 (NR1) subunit is decreased in hippocampal tissue from hAGS
and that the NMDAR component of Glu-induced increase in [Ca(2+)](i) is decreased in hippocampal slices from hAGS. Moreover,
the fraction of NR1 in the functional membrane pool in AGS is less than that in rats.
How inflammation promotes regeneration
Nat Neurosci. 2006 Jun; 9(6): 715-7.
No abstract available.
Ford G, Xu Z, Gates A, Jiang J, Ford BD
Expression Analysis Systematic Explorer (EASE) analysis reveals differential gene expression in permanent and transient focal
stroke rat models
Brain Res. 2006 Feb 3; 1071(1): 226-36.
To gain greater insight on the molecular mechanisms that underlie ischemic stroke, we compared gene expression profiles in
transient (tMCAO) and permanent middle cerebral artery occlusion (pMCAO) stroke models using Expression Analysis Systematic
Explorer (EASE) pathway analysis software. Many transcripts were induced in both stroke models, including genes associated
with transcriptional pathways, cell death, stress responses and metabolism. However, EASE analysis of the regulated genes
indicated molecular functions and biological processes unique to each model. Pathways associated with tMCAO included inflammation,
apoptosis and cell cycle, while pMCAO was associated with the induction of genes encoding neurotransmitter receptors, ion
channels, growth factors and signaling molecules. An intriguing finding was the involvement of tyrosine kinases and phosphatases
following pMCAO. These results provide evidence that neuronal death following tMCAO and pMCAO involves distinct mechanisms.
These findings may give new insight to the molecular mechanisms involved in stroke and may lead to novel neuroprotective strategies.
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